2651 - 2660 of 6433 Results
Title
Year
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OPENTitle: Cortical structural involvement and cognitive dysfunction in early Parkinson's diseaseJournal Name: NMR in BiomedicinePublisher: WileyVol: 31Issue #: 4Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/nbm.3900Citation Count: 8
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OPENTitle: Minimal Clinically Important Difference for UPDRS‐III in Daily PracticeJournal Name: Movement Disorders Clinical PracticePublisher: WileyVol: 5Issue #: 4Start Page: 448End Page: 450Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/mdc3.12632Best OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1002/mdc3.12632Citation Count: 46
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OPENTitle: Differential LRRK2 expression in the cortex, striatum, and substantia nigra in transgenic and nontransgenic rodentsJournal Name: Journal of Comparative NeurologyPublisher: WileyVol: 522Issue #: 11Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/cne.23612Best OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1002/cne.23612Citation Count: 4
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OPENTitle: Pharmacological antagonism of kainate receptor rescues dysfunction and loss of dopamine neurons in a mouse model of human parkin-induced toxicityJournal Name: Cell Death & DiseasePublisher: Springer Science and Business Media LLCVol: 11Issue #: 11Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41419-020-03172-8Best OA location URL: https://www.nature.com/articles/s41419-020-03172-8.pdfCitation Count: 20
- LRRK2 kinase inhibition reverses G2019S mutation-dependent effects on tau pathology progression2024OPENTitle: LRRK2 kinase inhibition reverses G2019S mutation-dependent effects on tau pathology progressionJournal Name: Translational NeurodegenerationPublisher: Springer Science and Business Media LLCVol: 13Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1186/s40035-024-00403-2Best OA location URL: https://translationalneurodegeneration.biomedcentral.com/counter/pdf/10.1186/s40035-024-00403-2Citation Count: 10
- Data-Driven Sequence of Changes to Anatomical Brain Connectivity in Sporadic Alzheimer’s Disease2017OPENTitle: Data-Driven Sequence of Changes to Anatomical Brain Connectivity in Sporadic Alzheimer’s DiseaseJournal Name: Frontiers in NeurologyPublisher: Frontiers Media SAVol: 8Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.3389/fneur.2017.00580Best OA location URL: https://www.frontiersin.org/articles/10.3389/fneur.2017.00580/pdfCitation Count: 49
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OPENTitle: Endoplasmic Reticulum Membrane Contact Sites, Lipid Transport, and NeurodegenerationJournal Name: Cold Spring Harbor Perspectives in BiologyPublisher: Cold Spring Harbor LaboratoryVol: 15Issue #: 4Start Page: a041257End Page: a041257Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1101/cshperspect.a041257Best OA location URL: https://pmc.ncbi.nlm.nih.gov/articles/PMC10071438/pdf/cshperspect-ERT-a041257.pdfCitation Count: 35
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OPENTitle: UCP2 mediates ghrelin’s action on NPY/AgRP neurons by lowering free radicalsJournal Name: NaturePublisher: Springer Science and Business Media LLCVol: 454Issue #: 7206Start Page: 846End Page: 851Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nature07181Citation Count: 685
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OPENTitle: Pitx3 is required for development of substantia nigra dopaminergic neuronsJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 100Issue #: 7Start Page: 4245End Page: 4250Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.0230529100Best OA location URL: http://doi.org/10.1073/pnas.0230529100Citation Count: 382
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OPENTitle: Compounds from an unbiased chemical screen reverse both ER-to-Golgi trafficking defects and mitochondrial dysfunction in Parkinson’s disease modelsJournal Name: Disease Models & MechanismsPublisher: The Company of BiologistsVol: 3Issue #: 3-4Start Page: 194End Page: 208Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1242/dmm.004267Best OA location URL: http://doi.org/10.1242/dmm.004267Citation Count: 178