3921 - 3930 of 6420 Results
Title
Year
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OPENTitle: Unified AI framework to uncover deep interrelationships between gene expression and Alzheimer’s disease neuropathologiesJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 12Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41467-021-25680-7Best OA location URL: https://www.nature.com/articles/s41467-021-25680-7.pdfCitation Count: 15
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OPENTitle: Atlas of RNA editing events affecting protein expression in aged and Alzheimer’s disease human brain tissueJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 12Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41467-021-27204-9Best OA location URL: https://escholarship.org/uc/item/0nc5b6htCitation Count: 42
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OPENTitle: Dysregulation of mitochondrial and proteolysosomal genes in Parkinson’s disease myeloid cellsJournal Name: Nature AgingPublisher: Springer Science and Business Media LLCVol: 1Issue #: 9Start Page: 850End Page: 863Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/s43587-021-00110-xBest OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/8728893Citation Count: 35
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OPENTitle: Disruption of mitochondrial complex I induces progressive parkinsonismJournal Name: NaturePublisher: Springer Science and Business Media LLCVol: 599Issue #: 7886Start Page: 650End Page: 656Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/s41586-021-04059-0Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/9189968Citation Count: 429
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OPENTitle: Genome sequencing analysis identifies new loci associated with Lewy body dementia and provides insights into its genetic architectureJournal Name: Nature GeneticsPublisher: Springer Science and Business Media LLCVol: 53Issue #: 3Start Page: 294End Page: 303Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/s41588-021-00785-3Best OA location URL: https://escholarship.org/uc/item/7qn6v913Citation Count: 314
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OPENTitle: Activation mechanism of PINK1Journal Name: NaturePublisher: Springer Science and Business Media LLCVol: 602Issue #: 7896Start Page: 328End Page: 335Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/s41586-021-04340-2Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/8828467Citation Count: 88
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OPENTitle: SETD7-mediated monomethylation is enriched on soluble Tau in Alzheimer’s diseaseJournal Name: Molecular NeurodegenerationPublisher: Springer Science and Business Media LLCVol: 16Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s13024-021-00468-xBest OA location URL: https://molecularneurodegeneration.biomedcentral.com/counter/pdf/10.1186/s13024-021-00468-xCitation Count: 21
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OPENTitle: Dual monoamine oxidase B and acetylcholine esterase inhibitors for treating movement and cognition deficits in a C. elegans model of Parkinson’s diseaseJournal Name: Medicinal Chemistry ResearchPublisher: Springer Science and Business Media LLCVol: 30Issue #: 5Start Page: 1166End Page: 1174Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1007/s00044-021-02720-xBest OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/8570587Citation Count: 4
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OPENTitle: APOE genotype dependent molecular abnormalities in the cerebrovasculature of Alzheimer’s disease and age-matched non-demented brainsJournal Name: Molecular BrainPublisher: Springer Science and Business Media LLCVol: 14Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s13041-021-00803-9Best OA location URL: https://molecularbrain.biomedcentral.com/counter/pdf/10.1186/s13041-021-00803-9Citation Count: 24
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OPENTitle: Endosomal traffic and glutamate synapse activity are increased in VPS35 D620N mutant knock-in mouse neurons, and resistant to LRRK2 kinase inhibitionJournal Name: Molecular BrainPublisher: Springer Science and Business Media LLCVol: 14Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s13041-021-00848-wBest OA location URL: https://molecularbrain.biomedcentral.com/track/pdf/10.1186/s13041-021-00848-wCitation Count: 17