4421 - 4430 of 8808 Results
Title
Year
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OPENTitle: A growth-factor-activated lysosomal K+ channel regulates Parkinson’s pathologyJournal Name: NaturePublisher: Springer Science and Business Media LLCVol: 591Issue #: 7850Start Page: 431End Page: 437Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/s41586-021-03185-zBest OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/7979525Citation Count: 98
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OPENTitle: Cautionary optimism: caffeine and Parkinson’s disease riskJournal Name: Journal of Clinical Movement DisordersPublisher: Springer Science and Business Media LLCVol: 3Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s40734-016-0037-8Best OA location URL: https://clinicalmovementdisorders.biomedcentral.com/track/pdf/10.1186/s40734-016-0037-8Citation Count: 5
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OPENTitle: Evaluation of rotigotine transdermal patch for the treatment of apathy and motor symptoms in Parkinson’s diseaseJournal Name: BMC NeurologyPublisher: Springer Science and Business Media LLCVol: 16Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1186/s12883-016-0610-7Best OA location URL: https://bmcneurol.biomedcentral.com/track/pdf/10.1186/s12883-016-0610-7Citation Count: 55
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OPENTitle: Intraventricular Sialidase Administration Enhances GM1 Ganglioside Expression and Is Partially Neuroprotective in a Mouse Model of Parkinson’s DiseaseJournal Name: PLOS ONEPublisher: Public Library of Science (PLoS)Vol: 10Issue #: 12Start Page: e0143351End Page: e0143351Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1371/journal.pone.0143351Best OA location URL: https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0143351&type=printableCitation Count: 34
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OPENTitle: Blood Biomarkers Associated with Cognitive Decline in Early Stage and Drug-Naive Parkinson’s Disease PatientsJournal Name: PLOS ONEPublisher: Public Library of Science (PLoS)Vol: 10Issue #: 11Start Page: e0142582End Page: e0142582Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1371/journal.pone.0142582Best OA location URL: https://journals.plos.org/plosone/article/file?id=10.1371/journal.pone.0142582&type=printableCitation Count: 52
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OPENTitle: Quantitative Measurement of Intact Alpha-Synuclein Proteoforms from Post-Mortem Control and Parkinson's Disease Brain Tissue by Intact Protein Mass SpectrometryJournal Name: Scientific ReportsPublisher: Springer Science and Business Media LLCVol: 4Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-saDOI - Digital Object Identifier: 10.1038/srep05797Best OA location URL: https://www.nature.com/articles/srep05797.pdfCitation Count: 155
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OPENTitle: Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer‐induced AMPA glutamate receptor signaling deficitsJournal Name: European Journal of NeurosciencePublisher: WileyVol: 39Issue #: 7Start Page: 1214End Page: 1224Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1111/ejn.12507Citation Count: 119
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OPENTitle: Impairment of PARK14-dependent Ca2+ signalling is a novel determinant of Parkinson’s diseaseJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 7Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/ncomms10332Best OA location URL: https://www.nature.com/articles/ncomms10332.pdfCitation Count: 92
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OPENTitle: Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiationJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 6Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/ncomms8314Best OA location URL: https://www.nature.com/articles/ncomms8314.pdfCitation Count: 280
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OPENTitle: XBP-1 deficiency in the nervous system protects against amyotrophic lateral sclerosis by increasing autophagyJournal Name: Genes & DevelopmentPublisher: Cold Spring Harbor LaboratoryVol: 23Issue #: 19Start Page: 2294End Page: 2306Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1101/gad.1830709Best OA location URL: http://genesdev.cshlp.org/content/23/19/2294.full.pdfCitation Count: 509