3791 - 3800 of 6422 Results
Title
Year
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OPENTitle: Evaluation of α-synuclein immunohistochemical methods used by invited expertsJournal Name: Acta NeuropathologicaPublisher: Springer Science and Business Media LLCVol: 116Issue #: 3Start Page: 277End Page: 288Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1007/s00401-008-0409-8Best OA location URL: http://doi.org/10.1007/s00401-008-0409-8Citation Count: 174
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OPENTitle: A novel transferrin/TfR2-mediated mitochondrial iron transport system is disrupted in Parkinson's diseaseJournal Name: Neurobiology of DiseasePublisher: Elsevier BVVol: 34Issue #: 3Start Page: 417End Page: 431Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.nbd.2009.02.009Best OA location URL: http://doi.org/10.1016/j.nbd.2009.02.009Citation Count: 189
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OPENTitle: High-Resolution Whole-Genome Association Study of Parkinson DiseaseJournal Name: The American Journal of Human GeneticsPublisher: Elsevier BVVol: 77Issue #: 5Start Page: 685End Page: 693Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1086/496902Best OA location URL: http://www.cell.com/article/S0002929707633540/pdfCitation Count: 510
- α-Synuclein, a chemoattractant, directs microglial migration via H2O2-dependent Lyn phosphorylation2015OPENTitle: α-Synuclein, a chemoattractant, directs microglial migration via H2O2-dependent Lyn phosphorylationJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 112Issue #: 15Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.1417883112Best OA location URL: https://www.pnas.org/content/pnas/112/15/E1926.full.pdfCitation Count: 139
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OPENTitle: Bid-mediated mitochondrial damage is a key mechanism in glutamate-induced oxidative stress and AIF-dependent cell death in immortalized HT-22 hippocampal neuronsJournal Name: Cell Death & DifferentiationPublisher: Springer Science and Business Media LLCVol: 18Issue #: 2Start Page: 282End Page: 292Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/cdd.2010.92Best OA location URL: https://www.nature.com/articles/cdd201092.pdfCitation Count: 179
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OPENTitle: Caspase-1 causes truncation and aggregation of the Parkinson’s disease-associated protein α-synucleinJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 113Issue #: 34Start Page: 9587End Page: 9592Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1073/pnas.1610099113Best OA location URL: https://www.pnas.org/content/pnas/113/34/9587.full.pdfCitation Count: 238
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OPENTitle: Data imputation and compression for Parkinson's disease clinical questionnairesJournal Name: Artificial Intelligence in MedicinePublisher: Elsevier BVVol: 114Issue #:Start Page: 102051End Page: 102051Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.artmed.2021.102051Best OA location URL: https://hal.science/hal-02570967/documentCitation Count: 23
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OPENTitle: Subtle anomaly detection: Application to brain MRI analysis of de novo Parkinsonian patientsJournal Name: Artificial Intelligence in MedicinePublisher: Elsevier BVVol: 125Issue #:Start Page: 102251End Page: 102251Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1016/j.artmed.2022.102251Best OA location URL: https://doi.org/10.1016/j.artmed.2022.102251Citation Count: 11
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OPENTitle: Changes in CD163+, CD11b+, and CCR2+ peripheral monocytes relate to Parkinson’s disease and cognitionJournal Name: Brain, Behavior, and ImmunityPublisher: Elsevier BVVol: 101Issue #:Start Page: 182End Page: 193Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1016/j.bbi.2022.01.005Best OA location URL: https://doi.org/10.1016/j.bbi.2022.01.005Citation Count: 43
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OPENTitle: The ADORA1 mutation linked to early-onset Parkinson’s disease alters adenosine A1-A2A receptor heteromer formation and functionJournal Name: Biomedicine & PharmacotherapyPublisher: Elsevier BVVol: 156Issue #:Start Page: 113896End Page: 113896Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1016/j.biopha.2022.113896Best OA location URL: https://doi.org/10.1016/j.biopha.2022.113896Citation Count: 11