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Year
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OPENTitle: Integrating brainstem and cortical functional architecturesJournal Name: Nature NeurosciencePublisher: Springer Science and Business Media LLCVol: 27Issue #: 12Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41593-024-01787-0Best OA location URL: https://doi.org/10.1038/s41593-024-01787-0Citation Count: 10
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RESTRICTEDTitle: A cross-disease resource of living human microglia identifies disease-enriched subsets and tool compounds recapitulating microglial statesJournal Name: Nature NeurosciencePublisher: Springer Science and Business Media LLCVol: 27Issue #: 12Start Page: 2521End Page: 2537Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1038/s41593-024-01764-7Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/12094270Citation Count: 9
- α-Synuclein aggregation decreases cortico-amygdala connectivity and impairs social behavior in mice2024OPENTitle: α-Synuclein aggregation decreases cortico-amygdala connectivity and impairs social behavior in miceJournal Name: Neurobiology of DiseasePublisher: Elsevier BVVol: 202Issue #:Start Page: 106702End Page: 106702Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1016/j.nbd.2024.106702Best OA location URL: https://doi.org/10.1016/j.nbd.2024.106702Citation Count: 2
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OPENTitle: Modelling human neuronal catecholaminergic pigmentation in rodents recapitulates age-related neurodegenerative deficitsJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 15Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41467-024-53168-7Best OA location URL: https://doi.org/10.1038/s41467-024-53168-7Citation Count: 2
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OPENTitle: Neuronal BAG3 attenuates tau hyperphosphorylation, synaptic dysfunction, and cognitive deficits induced by traumatic brain injury via the regulation of autophagy-lysosome pathwayJournal Name: Acta NeuropathologicaPublisher: Springer Science and Business Media LLCVol: 148Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1007/s00401-024-02810-1Citation Count: 2
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OPENTitle: High-throughput screening for small-molecule stabilizers of misfolded glucocerebrosidase in Gaucher disease and Parkinson’s diseaseJournal Name: Proceedings of the National Academy of SciencesPublisher: Proceedings of the National Academy of SciencesVol: 121Issue #: 42Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1073/pnas.2406009121Best OA location URL: https://doi.org/10.1073/pnas.2406009121Citation Count: 2
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OPENTitle: Single-cell somatic copy number variants in brain using different amplification methods and reference genomesJournal Name: Communications BiologyPublisher: Springer Science and Business Media LLCVol: 7Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1038/s42003-024-06940-wCitation Count: 1
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RESTRICTEDTitle: Somatic instability of the FGF14-SCA27B GAA•TTC repeat reveals a marked expansion bias in the cerebellumJournal Name: BrainPublisher: Oxford University Press (OUP)Vol: 148Issue #: 4Start Page:End Page:Publication Date:Open Access(OA) Status: RESTRICTEDLicense: cc-by, cc-by, cc-byDOI - Digital Object Identifier: 10.1093/brain/awae312Citation Count: 1
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OPENTitle: Sustained effect of prasinezumab on Parkinson’s disease motor progression in the open-label extension of the PASADENA trialJournal Name: Nature MedicinePublisher: Springer Science and Business Media LLCVol: 30Issue #: 12Start Page: 3669End Page: 3675Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41591-024-03270-6Best OA location URL: https://doi.org/10.1038/s41591-024-03270-6Citation Count: 11
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OPENTitle: α-Synuclein pathology disrupts mitochondrial function in dopaminergic and cholinergic neurons at-risk in Parkinson’s diseaseJournal Name: Molecular NeurodegenerationPublisher: Springer Science and Business Media LLCVol: 19Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s13024-024-00756-2Best OA location URL: https://doi.org/10.1186/s13024-024-00756-2Citation Count: 9