1211 - 1220 of 8571 Results
Title
Year
- Cortical Lewy bodies and Aβ burden are associated with prevalence and timing of dementia in Lewy body diseases2015OPENTitle: Cortical Lewy bodies and Aβ burden are associated with prevalence and timing of dementia in Lewy body diseasesJournal Name: Neuropathology and Applied NeurobiologyPublisher: WileyVol: 42Issue #: 5Start Page: 436End Page: 450Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1111/nan.12294Citation Count: 85
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OPENTitle: Visual Hallucinations in Eye Disease and Lewy Body DiseaseJournal Name: The American Journal of Geriatric PsychiatryPublisher: Elsevier BVVol: 24Issue #: 5Start Page: 350End Page: 358Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.jagp.2015.10.007Best OA location URL: http://www.ajgponline.org/article/S1064748115002687/pdfCitation Count: 28
- Adjusting for Underrepresentation Reveals Widespread Underestimation of Parkinson's Disease Symptom Burden2023RESTRICTEDTitle: Adjusting for Underrepresentation Reveals Widespread Underestimation of Parkinson's Disease Symptom BurdenJournal Name: Movement DisordersPublisher: WileyVol: 38Issue #: 9Start Page: 1679End Page: 1687Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1002/mds.29507Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/10524668Citation Count: 5
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RESTRICTEDTitle: Correlations between CSF proteins and spontaneous neuronal activity in Parkinson’s diseaseJournal Name: Neuroscience LettersPublisher: Elsevier BVVol: 673Issue #:Start Page: 61End Page: 66Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.1016/j.neulet.2018.02.062Citation Count: 6
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OPENTitle: Complementary motivational roles of nigroaccumbens and nigrostriatal dopaminergic pathwaysJournal Name: Movement DisordersPublisher: WileyVol: 34Issue #: 1Start Page: 45End Page: 45Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/mds.27504Best OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1002/mds.27504Citation Count: 3
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RESTRICTEDTitle: Biomarkers in Parkinson‘s Disease: A Funder‘s PerspectiveJournal Name: Biomarkers in MedicinePublisher: Informa UK LimitedVol: 4Issue #: 5Start Page: 723End Page: 729Publication Date:Open Access(OA) Status: RESTRICTEDLicense:DOI - Digital Object Identifier: 10.2217/bmm.10.89Citation Count: 17
- Nanomicellar formulation of coenzyme Q10 (Ubisol-Q10) effectively blocks ongoing neurodegeneration in the mouse 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model: potential use as an adjuvant treatment in Parkinson's disease2014OPENTitle: Nanomicellar formulation of coenzyme Q10 (Ubisol-Q10) effectively blocks ongoing neurodegeneration in the mouse 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine model: potential use as an adjuvant treatment in Parkinson's diseaseJournal Name: Neurobiology of AgingPublisher: Elsevier BVVol: 35Issue #: 10Start Page: 2329End Page: 2346Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.neurobiolaging.2014.03.032Best OA location URL: http://doi.org/10.1016/j.neurobiolaging.2014.03.032Citation Count: 68
- Hsp104 antagonizes α-synuclein aggregation and reduces dopaminergic degeneration in a rat model of Parkinson disease2008OPENTitle: Hsp104 antagonizes α-synuclein aggregation and reduces dopaminergic degeneration in a rat model of Parkinson diseaseJournal Name: Journal of Clinical InvestigationPublisher: American Society for Clinical InvestigationVol: 118Issue #: 9Start Page: 3087End Page: 3097Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1172/jci35781Best OA location URL: http://www.jci.org/articles/view/35781/files/pdfCitation Count: 195
- Rapid iPSC inclusionopathy models shed light on formation, consequence, and molecular subtype of α-synuclein inclusions2024OPENTitle: Rapid iPSC inclusionopathy models shed light on formation, consequence, and molecular subtype of α-synuclein inclusionsJournal Name: NeuronPublisher: Elsevier BVVol: 112Issue #: 17Start Page: 2886End Page: 2909.e16Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-by, cc-byDOI - Digital Object Identifier: 10.1016/j.neuron.2024.06.002Best OA location URL: https://doi.org/10.1016/j.neuron.2024.06.002Citation Count: 11
- JNK2 and JNK3 combined are essential for apoptosis in dopamine neurons of the substantia nigra, but are not required for axon degeneration2008OPENTitle: JNK2 and JNK3 combined are essential for apoptosis in dopamine neurons of the substantia nigra, but are not required for axon degenerationJournal Name: Journal of NeurochemistryPublisher: WileyVol: 107Issue #: 6Start Page: 1578End Page: 1588Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1111/j.1471-4159.2008.05713.xBest OA location URL: https://onlinelibrary.wiley.com/doi/pdfdirect/10.1111/j.1471-4159.2008.05713.x?download=trueCitation Count: 79