6081 - 6090 of 8715 Results
Title
Year
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OPENTitle: Revisiting the specificity and ability of phospho-S129 antibodies to capture alpha-synuclein biochemical and pathological diversityJournal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 8Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41531-022-00388-7Best OA location URL: https://www.nature.com/articles/s41531-022-00388-7.pdfCitation Count: 43
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OPENTitle: The glycoprotein GPNMB attenuates astrocyte inflammatory responses through the CD44 receptorJournal Name: Journal of NeuroinflammationPublisher: Springer Science and Business Media LLCVol: 15Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s12974-018-1100-1Best OA location URL: https://jneuroinflammation.biomedcentral.com/track/pdf/10.1186/s12974-018-1100-1Citation Count: 148
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OPENTitle: Current and projected future economic burden of Parkinson’s disease in the U.S.Journal Name: npj Parkinson's DiseasePublisher: Springer Science and Business Media LLCVol: 6Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41531-020-0117-1Best OA location URL: https://escholarship.org/uc/item/6wh3b5bzCitation Count: 495
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OPENTitle: Plasma phosphorylated tau 217 and phosphorylated tau 181 as biomarkers in Alzheimer's disease and frontotemporal lobar degeneration: a retrospective diagnostic performance studyJournal Name: The Lancet NeurologyPublisher: Elsevier BVVol: 20Issue #: 9Start Page: 739End Page: 752Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/s1474-4422(21)00214-3Best OA location URL: https://research.vumc.nl/en/publications/c35ddb07-cc7c-4ca6-ad06-b6cd811d4b63Citation Count: 424
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OPENTitle: Alpha-synuclein aggregates are phosphatase resistantJournal Name: Acta Neuropathologica CommunicationsPublisher: Springer Science and Business Media LLCVol: 12Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1186/s40478-024-01785-0Best OA location URL: https://actaneurocomms.biomedcentral.com/counter/pdf/10.1186/s40478-024-01785-0Citation Count: 4
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OPENTitle: Transcriptional programs mediating neuronal toxicity and altered glial–neuronal signaling in aDrosophilaknock-in tauopathy modelJournal Name: Genome ResearchPublisher: Cold Spring Harbor LaboratoryVol:Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1101/gr.278576.123Best OA location URL: https://genome.cshlp.org/content/early/2024/04/09/gr.278576.123.full.pdfCitation Count: 5
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OPENTitle: Effects of Serine 129 Phosphorylation on α-Synuclein Aggregation, Membrane Association, and InternalizationJournal Name: Journal of Biological ChemistryPublisher: Elsevier BVVol: 291Issue #: 9Start Page: 4374End Page: 4385Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1074/jbc.m115.705095Best OA location URL: http://www.jbc.org/article/S0021925820435859/pdfCitation Count: 145
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OPENTitle: Post hoc analysis of the Exenatide‐PD trial—Factors that predict responseJournal Name: European Journal of NeurosciencePublisher: WileyVol: 49Issue #: 3Start Page: 410End Page: 421Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1111/ejn.14096Citation Count: 63
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OPENTitle: Characterization of intracellular elevation of glutathione (GSH) with glutathione monoethyl ester and GSH in brain and neuronal cultures: Relevance to Parkinson's diseaseJournal Name: Experimental NeurologyPublisher: Elsevier BVVol: 203Issue #: 2Start Page: 512End Page: 520Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.expneurol.2006.09.004Best OA location URL: http://doi.org/10.1016/j.expneurol.2006.09.004Citation Count: 97
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OPENTitle: Lysosome and Inflammatory Defects in GBA1‐Mutant Astrocytes Are Normalized by LRRK2 InhibitionJournal Name: Movement DisordersPublisher: WileyVol: 35Issue #: 5Start Page: 760End Page: 773Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/mds.27994Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/8167931Citation Count: 89