1701 - 1710 of 8785 Results
Title
Year
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OPENTitle: Associations of motor and neuropsychiatric symptoms with comorbidities in prodromal Parkinson’s diseaseJournal Name: Frontiers in Aging NeurosciencePublisher: Frontiers Media SAVol: 16Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.3389/fnagi.2024.1452766Best OA location URL: https://doi.org/10.3389/fnagi.2024.1452766Citation Count: 0
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OPENTitle: Genetic analysis implicates APOE, SNCA and suggests lysosomal dysfunction in the etiology of dementia with Lewy bodiesJournal Name: Human Molecular GeneticsPublisher: Oxford University Press (OUP)Vol: 23Issue #: 23Start Page: 6139End Page: 6146Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1093/hmg/ddu334Best OA location URL: http://doi.org/10.1093/hmg/ddu334Citation Count: 218
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OPENTitle: Mitochondrial division inhibitor-1 is neuroprotective in the A53T-α-synuclein rat model of Parkinson’s diseaseJournal Name: Scientific ReportsPublisher: Springer Science and Business Media LLCVol: 7Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/s41598-017-07181-0Best OA location URL: https://www.nature.com/articles/s41598-017-07181-0.pdfCitation Count: 115
- ATP13A2 Regulates Cellular α-Synuclein Multimerization, Membrane Association, and Externalization2021OPENTitle: ATP13A2 Regulates Cellular α-Synuclein Multimerization, Membrane Association, and ExternalizationJournal Name: International Journal of Molecular SciencesPublisher: MDPI AGVol: 22Issue #: 5Start Page: 2689End Page: 2689Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.3390/ijms22052689Best OA location URL: https://www.mdpi.com/1422-0067/22/5/2689/pdf?version=1615453355Citation Count: 23
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OPENTitle: Quantitative Measurement of Intact Alpha-Synuclein Proteoforms from Post-Mortem Control and Parkinson's Disease Brain Tissue by Intact Protein Mass SpectrometryJournal Name: Scientific ReportsPublisher: Springer Science and Business Media LLCVol: 4Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-saDOI - Digital Object Identifier: 10.1038/srep05797Best OA location URL: https://www.nature.com/articles/srep05797.pdfCitation Count: 155
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OPENTitle: Asparagine endopeptidase cleaves α-synuclein and mediates pathologic activities in Parkinson's diseaseJournal Name: Nature Structural & Molecular BiologyPublisher: Springer Science and Business Media LLCVol: 24Issue #: 8Start Page: 632End Page: 642Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1038/nsmb.3433Best OA location URL: https://www.nature.com/articles/nsmb.3433.pdfCitation Count: 169
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OPENTitle: Tau phosphorylation and tau mislocalization mediate soluble Aβ oligomer‐induced AMPA glutamate receptor signaling deficitsJournal Name: European Journal of NeurosciencePublisher: WileyVol: 39Issue #: 7Start Page: 1214End Page: 1224Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1111/ejn.12507Citation Count: 119
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OPENTitle: Mitochondrial Function in Hereditary Spastic Paraplegia: Deficits in SPG7 but Not SPAST Patient-Derived Stem CellsJournal Name: Frontiers in NeurosciencePublisher: Frontiers Media SAVol: 14Issue #:Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.3389/fnins.2020.00820Best OA location URL: https://www.frontiersin.org/articles/10.3389/fnins.2020.00820/pdfCitation Count: 26
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OPENTitle: Impairment of PARK14-dependent Ca2+ signalling is a novel determinant of Parkinson’s diseaseJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 7Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/ncomms10332Best OA location URL: https://www.nature.com/articles/ncomms10332.pdfCitation Count: 92
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OPENTitle: Parkinson-causing α-synuclein missense mutations shift native tetramers to monomers as a mechanism for disease initiationJournal Name: Nature CommunicationsPublisher: Springer Science and Business Media LLCVol: 6Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1038/ncomms8314Best OA location URL: https://www.nature.com/articles/ncomms8314.pdfCitation Count: 280