1311 - 1320 of 8571 Results
Title
Year
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OPENTitle: miR-34a as hub of T cell regulation networksJournal Name: Journal for ImmunoTherapy of CancerPublisher: BMJVol: 7Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s40425-019-0670-5Best OA location URL: https://jitc.bmj.com/content/jitc/7/1/187.full.pdfCitation Count: 46
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OPENTitle: Mitochondrial therapies for Parkinson's diseaseJournal Name: Movement DisordersPublisher: WileyVol: 25Issue #: S1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1002/mds.22781Best OA location URL: https://www.ncbi.nlm.nih.gov/pmc/articles/5797696Citation Count: 58
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OPENTitle: Deep Brain Stimulation associated gliosis: A post-mortem studyJournal Name: Parkinsonism & Related DisordersPublisher: Elsevier BVVol: 54Issue #:Start Page: 51End Page: 55Publication Date:Open Access(OA) Status: OPENLicense: other-oaDOI - Digital Object Identifier: 10.1016/j.parkreldis.2018.04.009Best OA location URL: https://doi.org/10.1016/j.parkreldis.2018.04.009Citation Count: 29
- LGALS3 (galectin 3) mediates an unconventional secretion of SNCA/α-synuclein in response to lysosomal membrane damage by the autophagic-lysosomal pathway in human midbrain dopamine neurons2021OPENTitle: LGALS3 (galectin 3) mediates an unconventional secretion of SNCA/α-synuclein in response to lysosomal membrane damage by the autophagic-lysosomal pathway in human midbrain dopamine neuronsJournal Name: AutophagyPublisher: Informa UK LimitedVol: 18Issue #: 5Start Page: 1020End Page: 1048Publication Date:Open Access(OA) Status: OPENLicense: cc-by-nc-ndDOI - Digital Object Identifier: 10.1080/15548627.2021.1967615Best OA location URL: https://www.tandfonline.com/doi/pdf/10.1080/15548627.2021.1967615?needAccess=trueCitation Count: 41
- Intracellular delivery of Parkin rescues neurons from accumulation of damaged mitochondria and pathological α-synuclein2020OPENTitle: Intracellular delivery of Parkin rescues neurons from accumulation of damaged mitochondria and pathological α-synucleinJournal Name: Science AdvancesPublisher: American Association for the Advancement of Science (AAAS)Vol: 6Issue #: 18Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-ncDOI - Digital Object Identifier: 10.1126/sciadv.aba1193Best OA location URL: https://advances.sciencemag.org/content/advances/6/18/eaba1193.full.pdfCitation Count: 58
- Pathological α-synuclein distribution in subjects with coincident Alzheimer’s and Lewy body pathology2015OPENTitle: Pathological α-synuclein distribution in subjects with coincident Alzheimer’s and Lewy body pathologyJournal Name: Acta NeuropathologicaPublisher: Springer Science and Business Media LLCVol: 131Issue #: 3Start Page: 393End Page: 409Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1007/s00401-015-1526-9Citation Count: 145
- Incorporating spatial constraint in co-activation pattern analysis to explore the dynamics of resting-state networks: An application to Parkinson's disease2018OPENTitle: Incorporating spatial constraint in co-activation pattern analysis to explore the dynamics of resting-state networks: An application to Parkinson's diseaseJournal Name: NeuroImagePublisher: Elsevier BVVol: 172Issue #:Start Page: 64End Page: 84Publication Date:Open Access(OA) Status: OPENLicense:DOI - Digital Object Identifier: 10.1016/j.neuroimage.2018.01.019Citation Count: 35
- Longitudinal tracking of neuronal mitochondria delineates PINK1/Parkin-dependent mechanisms of mitochondrial recycling and degradation2021OPENTitle: Longitudinal tracking of neuronal mitochondria delineates PINK1/Parkin-dependent mechanisms of mitochondrial recycling and degradationJournal Name: Science AdvancesPublisher: American Association for the Advancement of Science (AAAS)Vol: 7Issue #: 32Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by-ncDOI - Digital Object Identifier: 10.1126/sciadv.abf6580Best OA location URL: https://www.science.org/doi/pdf/10.1126/sciadv.abf6580Citation Count: 28
- Leucine-rich repeat kinase 2 controls protein kinase A activation state through phosphodiesterase 42018OPENTitle: Leucine-rich repeat kinase 2 controls protein kinase A activation state through phosphodiesterase 4Journal Name: Journal of NeuroinflammationPublisher: Springer Science and Business Media LLCVol: 15Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-byDOI - Digital Object Identifier: 10.1186/s12974-018-1337-8Best OA location URL: https://jneuroinflammation.biomedcentral.com/track/pdf/10.1186/s12974-018-1337-8Citation Count: 42
- Pathogenic LRRK2 variants are gain-of-function mutations that enhance LRRK2-mediated repression of β-catenin signaling2017OPENTitle: Pathogenic LRRK2 variants are gain-of-function mutations that enhance LRRK2-mediated repression of β-catenin signalingJournal Name: Molecular NeurodegenerationPublisher: Springer Science and Business Media LLCVol: 12Issue #: 1Start Page:End Page:Publication Date:Open Access(OA) Status: OPENLicense: cc-by, cc-byDOI - Digital Object Identifier: 10.1186/s13024-017-0153-4Best OA location URL: https://molecularneurodegeneration.biomedcentral.com/track/pdf/10.1186/s13024-017-0153-4Citation Count: 51